Marie J. Stuart, MD

Contact Dr. Stuart

1025 Walnut Street
College Building, Room 727h
Philadelphia, PA 19107

(215) 955-9820
(215) 955-8011 fax

Most Recent Peer-reviewed Publications

  1. Pain characteristics and age-related pain trajectories in infants and young children with sickle cell disease
  2. Tissue factor-positive monocytes in children with sickle cell disease: Correlation with biomarkers of haemolysis
  3. Tapered oral dexamethasone for the acute chest syndrome of sickle cell disease
  4. Increased levels of the inflammatory biomarker C-reactive protein at baseline are associated with childhood sickle cell vasocclusive crises
  5. Heme induces endothelial tissue factor expression: Potential role in hemostatic activation in patients with hemolytic anemia
  6. Increased von Willebrand factor antigen and high molecular weight multimers in sickle cell disease associated with nocturnal hypoxemia
  7. Vaso-occlusion in children with sickle cell disease: Clinical characteristics and biologic correlates
  8. Sickle-cell disease
  9. Hypoxaemia in sickle cell disease: Biomarker modulation and relevance to pathophysiology
  10. Role of erythrocyte phosphatidylserine in sickle red cell-endothelial adhesion
  11. Eicosanoids in sickle cell disease: Potential relevance of neutrophil leukotriene B4to disease pathophysiology
  12. Thrombophilia in sickle cell disease: The red cell connection
  13. Fetal hemoglobin in sickle cell anemia: Relationship to erythrocyte adhesion markers and adhesion
  14. Acute chest syndrome of sickle cell disease: New light on an old problem
  15. Hemostatic alterations in sickle cell disease: Relationships to disease pathophysiology
  16. Fetal hemoglobin in sickle cell disease: Relationship to erythrocyte phosphatidylserine exposure and coagulation activation
  17. Sickle cell acute chest syndrome: Pathogenesis and rationale for treatment
  18. Eicosanoids in sickle cell disease: Potential relevance of 12(S)-hydroxy-5,8,10,14-eicosatetraenoic acid to the pathophysiology of vaso-occlusion
  19. Vascular cell adhesion molecule-1 is involved in mediating hypoxia- induced sickle red blood cell adherence to endothelium: Potential role in sickle cell disease
  20. Impaired mobilization of intracellular calcium in neonatal platelets