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Linda D. Siracusa, PhD

Contact Dr. Siracusa

233 South Tenth Street
Bluemle Life Sciences Building, Room 719
Philadelphia, PA 19107-5541

(215) 503-4536
(215) 923-4153 fax

Most Recent Peer-reviewed Publications

  1. Understanding mitochondrial polymorphisms in cancer
  2. The Clock Is Ticking: Countdown to Metastases
  3. The 28th International Mammalian Genome Conference—meeting report
  4. Collagen content in skin and internal organs of the Tight Skin mouse: An animal model of scleroderma
  5. Identification of five novel modifier loci of ApcMinharbored in the BXH14 recombinant inbred strain
  6. Tpl2 ablation promotes intestinal inflammation and tumorigenesis in Apcminmice by inhibiting IL-10 secretion and regulatory T-cell generation
  7. Technical approaches for mouse models of human disease
  8. Identification of Mom12 and Mom13, two novel modifier loci of ApcMin-mediated intestinal tumorigenesis
  9. The armadillo repeat domain of Apc suppresses intestinal tumorigenesis
  10. A Tcf4-GFP reporter mouse model for monitoring effects of Apc mutations during intestinal tumorigenesis
  11. The noncoding RNAs: A genomic symphony of transcripts
  12. Cancer-associated genomic regions (CAGRs) and noncoding RNAs: Bioinformatics and therapeutic implications
  13. Influence of a nonfragile FHIT transgene on murine tumor susceptibility
  14. Guanylyl Cyclase C Suppresses Intestinal Tumorigenesis by Restricting Proliferation and Maintaining Genomic Integrity
  15. MicroRNA genes are frequently located near mouse cancer susceptibility loci
  16. The modifier of Min 2 (Mom2) locus: Embryonic lethality of a mutation in the Atp5a1 gene suggests a novel mechanism of polyp suppression
  17. Mammalian microRNAs: A small world for fine-tuning gene expression
  18. A mutation in stratifin is responsible for the repeated epilation (Er) phenotype in mice
  19. Diversity in secreted PLA2-IIA activity among inbred mouse strains that are resistant or susceptible to ApcMin/+ tumorigenesis
  20. Analysis of reciprocal congenic lines reveals the C3H/HeJ genome to be highly resistant to ApcMinintestinal tumorigenesis