Linda D. Siracusa, PhD

Contact Dr. Siracusa

233 South Tenth Street
Bluemle Life Sciences Building, Room 719
Philadelphia, PA 19107-5541

(215) 503-4536
(215) 923-4153 fax

Most Recent Peer-reviewed Publications

  1. Collagen content in skin and internal organs of the Tight Skin mouse: An animal model of scleroderma
  2. Identification of five novel modifier loci of Apc Min harbored in the BXH14 recombinant inbred strain
  3. The armadillo repeat domain of Apc suppresses intestinal tumorigenesis
  4. A Tcf4-GFP reporter mouse model for monitoring effects of Apc mutations during intestinal tumorigenesis
  5. The noncoding RNAs: A genomic symphony of transcripts
  6. Cancer-associated genomic regions (CAGRs) and noncoding RNAs: Bioinformatics and therapeutic implications
  7. Influence of a nonfragile FHIT transgene on murine tumor susceptibility
  8. Guanylyl Cyclase C Suppresses Intestinal Tumorigenesis by Restricting Proliferation and Maintaining Genomic Integrity
  9. MicroRNA genes are frequently located near mouse cancer susceptibility loci
  10. The modifier of Min 2 (Mom2) locus: Embryonic lethality of a mutation in the Atp5a1 gene suggests a novel mechanism of polyp suppression
  11. Mammalian microRNAs: A small world for fine-tuning gene expression
  12. A mutation in stratifin is responsible for the repeated epilation (Er) phenotype in mice
  13. Diversity in secreted PLA2-IIA activity among inbred mouse strains that are resistant or susceptible to ApcMin/+tumorigenesis
  14. Analysis of reciprocal congenic lines reveals the C3H/HeJ genome to be highly resistant to Apc Min intestinal tumorigenesis
  15. The Collaborative Cross, a community resource for the genetic analysis of complex traits
  16. Skeletal dysplasia and male infertility locus on mouse chromosome 9
  17. A role for the androgen receptor in collagen content of the skin
  18. The nature and identification of quantitative trait loci: A community's view
  19. The cohesin SMC3 is a target the for β-catenin/TCF4 transactivation pathway
  20. Nonneuronal expression of the GABAA β3 subunit gene is required for normal palate development in mice